Search results for "adaptive immune system"

showing 4 items of 4 documents

Loss of histone macroH2A1 in hepatocellular carcinoma cells promotes paracrine-mediated chemoresistance and CD4

2019

Rationale: Loss of histone macroH2A1 induces appearance of cancer stem cells (CSCs)-like cells in hepatocellular carcinoma (HCC). How CSCs interact with the tumor microenvironment and the adaptive immune system is unclear. Methods: We screened aggressive human HCC for macroH2A1 and CD44 CSC marker expression. We also knocked down (KD) macroH2A1 in HCC cells, and performed integrated transcriptomic and secretomic analyses. Results: Human HCC showed low macroH2A1 and high CD44 expression compared to control tissues. MacroH2A1 KD CSC-like cells transferred paracrinally their chemoresistant properties to parental HCC cells. MacroH2A1 KD conditioned media transcriptionally reprogrammed parental …

MaleCarcinoma HepatocellularT-Lymphocytes RegulatoryHistonesadaptive immune systemCell Line TumorParacrine CommunicationTumor MicroenvironmentHumansMetabolomicschemoresistance.neoplasmsLiver Neoplasmshistone macroH2A1Interleukin-2 Receptor alpha SubunitForkhead Transcription Factorshepatocellular carcinomaMiddle Ageddigestive system diseasesGene Expression Regulation NeoplasticHyaluronan ReceptorsDrug Resistance NeoplasmGene Knockdown TechniquesNeoplastic Stem CellsGlycolysisResearch PaperTheranostics
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Introduction. Ecological immunology.

2009

12 pages; International audience; An organism's fitness is critically reliant on its immune system to provide protection against parasites and pathogens. The structure of even simple immune systems is surprisingly complex and clearly will have been moulded by the organism's ecology. The aim of this review and the theme issue is to examine the role of different ecological factors on the evolution of immunity. Here, we will provide a general framework of the field by contextualizing the main ecological factors, including interactions with parasites, other types of biotic as well as abiotic interactions, intraspecific selective constraints (life-history trade-offs, sexual selection) and popula…

[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyEcology (disciplines)Populationinnate immune systemecological immunologyBiology[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyGeneral Biochemistry Genetics and Molecular BiologyImmune systemadaptive immune systemMESH : Ecosystemmicrobiota[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisMESH : EvolutioneducationMESH : Host-Pathogen InteractionsOrganismCoevolutiontrade-offIntroductioneducation.field_of_study[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyInnate immune systemResistance (ecology)EcologyMESH : HumansAcquired immune systemMESH : Genetics PopulationMESH : ImmunitycoevolutionMESH : AnimalsGeneral Agricultural and Biological Sciences
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Loss of histone macroH2A1 in hepatocellular carcinoma cells promotes paracrine-mediated chemoresistance and CD4+CD25+FoxP3+ regulatory T cells activa…

2020

Rationale: Loss of histone macroH2A1 induces appearance of cancer stem cells (CSCs)-like cells in hepatocellular carcinoma (HCC). How CSCs interact with the tumor microenvironment and the adaptive immune system is unclear. Methods: We screened aggressive human HCC for macroH2A1 and CD44 CSC marker expression. We also knocked down (KD) macroH2A1 in HCC cells, and performed integrated transcriptomic and secretomic analyses. Results: Human HCC showed low macroH2A1 and high CD44 expression compared to control tissues. MacroH2A1 KD CSC-like cells transferred paracrinally their chemoresistant properties to parental HCC cells. MacroH2A1 KD conditioned media transcriptionally reprogrammed parental …

EXPRESSION0301 basic medicineLIVERAdaptive immune systemPOSTTRANSCRIPTIONAL CONTROLHepatocellular carcinomaMedicine (miscellaneous)PROGRESSIONHistone macroH2A1Research & Experimental MedicineCONTRIBUTES03 medical and health sciencesParacrine signalling0302 clinical medicineadaptive immune systemCancer stem cellCANCER STEM-CELLSmedicinechemoresistance.TRANSCRIPTIONIL-2 receptorneoplasmsPharmacology Toxicology and Pharmaceutics (miscellaneous)Tumor microenvironmentScience & Technologybiologyhistone macroH2A1CD44PROLIFERATIONchemoresistanceCancerFOXP3hepatocellular carcinomamedicine.diseaseAcquired immune systemdigestive system diseases3. Good healthCYTOKINE030104 developmental biologyMedicine Research & ExperimentalSENESCENCE030220 oncology & carcinogenesisCancer researchbiology.proteinLife Sciences & BiomedicineChemoresistance
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Gut dysbiosis and adaptive immune response in diet-induced obesity vs. Systemic inflammation

2017

A mutual interplay exists between adaptive immune system and gut microbiota. Altered gut microbial ecosystems are associated with the metabolic syndrome, occurring in most obese individuals. However, it is unknown why 10-25% of obese individuals are metabolically healthy, while normal weight individuals can develop inflammation and atherosclerosis. We modelled these specific metabolic conditions in mice fed with a chow diet, an obesogenic but not inflammatory diet - mimicking healthy obesity, or Paigen diet - mimicking inflammation in the lean subjects. We analysed a range of markers and cytokines in the aorta, heart, abdominal fat, liver and spleen, and metagenomics analyses were performed…

0301 basic medicineMicrobiology (medical)medicine.medical_specialtyAdaptive immune systemlcsh:QR1-502SpleenInflammationGut microbiotaGut floraSystemic inflammationMicrobiologylcsh:Microbiology03 medical and health sciencesImmune systemInternal medicineErysipelotrichiamedicineObesityOriginal ResearchInflammationbiologymedicine.diseaseAcquired immune systembiology.organism_classification030104 developmental biologymedicine.anatomical_structureEndocrinologyAdaptive immune system; Gut microbiota; Inflammation; Obesity; Microbiology; Microbiology (medical)ImmunologyMetabolic syndromemedicine.symptom
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